Determine etiology and precipitating factors (spontaneous collapse, trauma, malignancy, infection, complication of mechanical ventilation). |
Understanding the cause of lung collapse is necessary for proper chest tube placement and choice of other therapeutic measures. |
Check out respiratory function, noting rapid or shallow respirations, dyspnea, reports of “air hunger,” development of cyanosis, changes in vital signs. |
Respiratory distress and changes in vital signs may occur as a result of physiological stress and pain or may indicate development of shock due to hypoxia or hemorrhage. |
Observe for synchronous respiratory pattern when using mechanical ventilator. Note changes in airway pressures. |
Difficulty breathing “with” ventilator and increasing airway pressures suggests worsening of condition or development of complications (spontaneous rupture of a bleb creating a new pneumo -thorax). |
Auscultate breath sounds. |
Breath sounds may be diminished or absent in a lobe, lung segment, or entire lung field (unilateral). Atelectatic area will have no breath sounds, and partially collapsed areas have decreased sounds. Regularly scheduled evaluation also helps determine areas of good air exchange and provides a baseline to evaluate resolution of pneumothorax. |
Note chest excursion and position of trachea. |
Chest excursion is unequal until lung re-expands. Trachea deviates away from affected side with tension pneumothorax. |
Evaluate fremitus. |
Voice and tactile fremitus (vibration) is reduced in fluid-filled or consolidated tissue. |
Assist patient with splinting painful area when coughing, deep breathing. |
Supporting chest and abdominal muscles makes coughing more effective and less traumatic. |
Maintain position of comfort, usually with head of bed elevated. Turn to affected side. Encourage patient to sit up as much as possible. |
Promotes maximal inspiration; enhances lung expansion and ventilation in unaffected side. |
Maintain a calm attitude, assisting patient to “take control” by using slower and deeper respirations. |
Assists patient to deal with the physiological effects of hypoxia, which may be manifested as anxiety or fear. |
Once chest tube is inserted: |
Maintains prescribed intrapleural negativity, which promotes optimum lung expansion and fluid drainage. Note: Dry- seal setups are also used with an automatic control valve (AVC), which provides a one-way valve seal similar to that achieved with the water-seal system. |
Check suction control chamber for correct amount of suction (determined by water level, wall or table regulator at correct setting; |
Water in a sealed chamber serves as a barrier that prevents atmospheric air from entering the pleural space should the suction source be disconnected and aids in evaluating whether the chest drainage system is functioning appropriately. Note: Underfilling the water-seal chamber leaves it exposed to air, putting patient at risk for pneumothorax or tension pneumothorax. Overfilling (a more common mistake) prevents air from easily exiting the pleural space, thus preventing resolution of pneumothorax or tension pneumothorax. |
Monitor fluid level in water-seal chamber; maintain at prescribed level; |
Observe water-seal chamber bubbling; |
Bubbling during expiration reflects venting of pneumothorax (desired action). Bubbling usually decreases as the lung expands or may occur only during expiration or coughing as the pleural space diminishes. Absence of bubbling may indicate complete lung re-expansion (normal) or represent complications such as obstruction in the tube. |
Observe for abnormal and continuous water-seal chamber bubbling; |
With suction applied, this indicates a persistent air leak that may be from a large pneumothorax at the chest insertion site (patient-centered) or chest drainage unit (system-centered). |
Know the location of air leak (patient- or system-centered) by clamping thoracic catheter just distal to exit from chest; |
If bubbling stops when catheter is clamped at insertion site, leak is patient- centered (at insertion site or within the patient). |
Place petrolatum gauze and other appropriate material around the insertion as indicated. |
Usually corrects insertion site air leak. |
Clamp tubing in stepwise fashion downward toward drainage unit if air leak continues; |
Isolates location of a system-centered air leak.Note: Information indicates that clamping for a suspected leak may be the only time that chest tube should be clamped. |
Seal drainage tubing connection sites securely with lengthwise tape or bands according to established policy; |
Prevents and corrects air leaks at connector sites. |
Monitor water-seal chamber “tidaling.” Note whether change is transient or permanent; |
The water-seal chamber serves as an intrapleural manometer (gauges intrapleural pressure); therefore, fluctuation (tidaling) reflects pressure differences between inspiration and expiration. Tidaling of 2–6 cm during inspiration is normal and may increase briefly during coughing episodes. Continuation of excessive tidal fluctuations may indicate existence of airway obstruction or presence of a large pneumothorax. |
Position drainage system tubing for optimal function like shorten tubing or coil extra tubing on bed, making sure tubing is not kinked or hanging below entrance to drainage container. Drain accumulated fluid as necessary; |
Improper position, kinking, or accumulation of clots or fluid in the tubing changes the desired negative pressure and impedes air or fluid evacuation. Note: If a dependent loop in the drainage tube cannot be avoided, lifting and draining it every 15 min will maintain adequate drainage in the presence of a hemothorax. |
Assess amount of chest tube drainage, noting whether tube is warm and full of blood and bloody fluid level in water-seal bottle is rising; |
Useful in evaluating resolution of pneumothorax and development of hemorrhage requiring prompt intervention. Note: Some drainage systems are equipped with an autotransfusion device, which allows for salvage of shed blood. |
Evaluate need for tube stripping (“milking”); |
Although routine stripping is not recommended, it may be necessary occasionally to maintain drainage in the presence of fresh bleeding, large blood clots or purulent exudate (empyema). |
Strip tubes carefully per protocol, in a manner that minimizes excess negative pressure. |
Stripping is usually uncomfortable for patient because of the change in intrathoracic pressure, which may induce coughing or chest discomfort. Vigorous stripping can create very high intrathoracic suction pressure, which can be injurious (invagination of tissue into catheter eyelets, collapse of tissues around the catheter, and bleeding from rupture of small blood vessels). |
If thoracic catheter is disconnected or dislodged: |
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Observe for signs of respiratory distress. If possible, reconnect thoracic catheter to tubing or suction, using clean technique. If the catheter is dislodged from the chest, cover insertion site immediately with petrolatum dressing and apply firm pressure. Notify physician at once. |
Pneumothorax may recur, requiring prompt intervention to prevent fatal pulmonary and circulatory impairment. |
After thoracic catheter is removed: |
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Cover insertion site with sterile occlusive dressing. Observe for signs and symptoms that may indicate recurrence of pneumothorax (shortness of breath, reports of pain. Inspect insertion site, note character of drainage). |
Early detection of a developing complication is essential (recurrence of pneumothorax, presence of infection). |
Review serial chest x-rays. |
Monitors progress of resolving hemothorax or pneumothorax and re-expansion of lung. Can identify malposition of endotracheal tube (ET) affecting lung re-expansion. |
Monitor and graph serial ABGs and pulse oximetry. Review vital capacity and tidal volume measurements. |
Assesses status of gas exchange and ventilation, need for continuation or alterations in therapy. |
Administer supplemental oxygen via cannula, mask, or mechanical ventilation as indicated. |
Aids in reducing work of breathing; promotes relief of respiratory distress and cyanosis associated with hypoxemia. |